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. 2019 Apr 16;13:41. doi: 10.3389/fnana.2019.00041

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Copyright © 2019 Kanold, Deng and Meng.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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(A) SPNs show functional and morphological hyperconnectivity after hypoxic-ischemic (HI; from Sheikh et al., 2019; no copyright permission as required for use of this image). Shown are LSPS maps of connection probability and neurolucida reconstructions. (B) Cartoon illustrating how depolarization caused by HI can lead to unsilencing of synapses on SPNs (middle). If presynaptic cells are active (indicated by spike train), the coincidence between presynaptic activity and postsynaptic depolarization might result in a subsequent increase in AMPARs (right, black).