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. 2019 Apr 16;10:309. doi: 10.3389/fphys.2019.00309

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Copyright © 2019 Watson, Gould, Penfold, Lambert, Pratama, Dai, Gray, Head and Jandeleit-Dahm.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Working hypothesis relating to changes in catecholamines and oxidation in the kidney cortex of mice with hypertension and diabetes with respect to monoamine oxidase (MAO-A). Dotted lines represent theorized outcomes. (–): no change; ↑: increase in levels; ↑↑: large increase in levels; ↓: decrease in levels. PCT, proximal convoluted tubules; MAO, monoamine oxidase levels; sod2- superoxide dismutase 2 (MnSOD) gene transcript levels; TH, tyrosine hydroxylase protein levels. Large bottom arrows: relation to albuminuria and the overall level of kidney injury seen.