Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2019 Apr 10;20(7):1785. doi: 10.3390/ijms20071785

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2019 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

PMC Copyright notice

Effect of mitogen-activated protein kinases (MAPKs) on the mechanisms involved in hepatic ischemia-reperfusion injury. Ang: angiotensin; AP-1: activating protein; cAMP: cyclic adenosine monophosphate; Cyt c: cytochrome c; EC: endothelial cell; ERK 1/2: extracellular signal regulated kinases 1/2; ET: endothelin; GM-CSF: granulocyte-macrophage colony-stimulating factor; GSH: glutathione; HO-1: heme oxygenase-1; HSP72: heat shock protein 72; ICAM: intracellular cell adhesion molecule; IL: interleukin; JNK: c-jun N-terminal kinase; KC: kupffer cell; LTB4: leucotriene B4; NO: nitric oxide; ONOO-: peroxynitrite; PAF: platelet activating factor; PBK: protein kinase B; PI3K: phosphoinositide 3-kinase; RBP4: retinol binding protein 4; ROS: reactive oxygen species; SLPI: secretory leukocyte protease inhibitor; SOD: superoxide dismutase; TNF-α: tumor necrosis factor-alpha; VCAM: vascular cell adhesion molecule; X/XOD: xanthine/xanthine oxidase.