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. 2019 Apr 8;20(7):1737. doi: 10.3390/ijms20071737

Table 1.

Human pluripotent stem cell (hPSC) lines used in this work.

Cell Line Passage Karyotype/CGH Origin
iPSC lines
PB3 43 46, XX Normal amniocyte
PB4 26 46, XY Amniocyte, sickle cell anemia
PB6 23 47, XY, +21 Amniocyte, Down syndrome
PB6.1 14 47, XY, +21 Amniocyte, Down syndrome
PB7 63 46, XY Normal amniocyte
PB9 17 46, XY Normal amniocyte
PB10 16 46, XY Normal amniocyte
PB12.1 33 47, XY, +21 Amniocyte, Down syndrome
PB13 28 46, XY Endothelial progenitor cell from peripheral blood
PB14.3 41 47, XX, +21 Amniocyte, Down syndrome
PB17 38 46, XY Endothelial progenitor cell from peripheral blood
ESC lines
SA01 22+4+7 46, XY Cellartis AB, Sweden
H1 67 46, XY WiCell, Wisconsin, USA
H9 31 46, XX WiCell, Wisconsin, USA
CL01 16 46, XY, der21, t(1;21) Paris Sud University, France

All iPSC lines were generated in ESTeam Paris Sud University, Inserm U935, Villejuif, France. CGH, Comparative Genomic Hybridization; der21, t(1;21), unbalanced chromosome translocation involving chromosome 1 and chromosome 21.