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. 2019 Apr 25;19:391. doi: 10.1186/s12885-019-5605-5

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — PTEN inhibition leads to increased expression of EMT-associated genes expression in the absence of RBP-Jk. a Western blot analyses showing the protein level of PTEN, FAK, p-FAK and EMT-related genes (N-cadherin, E-cadherin, Snail-1, MMP-9) in NC (sh-NC), RBP-Jκ-knockdown (RBP-Jκ shRNA), RBP-Jκ-knockdown (RBP-Jκ shRNA) plus VO-Ohpic trihydrat-treated SCC-9 and CAL-27 cells. GAPDH acts as internal control. b Gray level analysis of the protein level of PTEN, FAK, p-FAK and EMT-related genes (N-cadherin, E-cadherin, Snail-1, MMP-9) in NC (sh-NC), RBP-Jκ-knockdown (RBP-Jκ shRNA), RBP-Jκ-knockdown (RBP-Jκ shRNA) plus VO-Ohpic trihydrat-treated SCC-9 and CAL-27 cells. The mean ± SD represents the average levels from 3 replicates. *p < 0.05, **p < 0.01