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. 2019 Apr 26;38:179. doi: 10.1186/s13046-019-1174-3

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — LINC01197 significantly inhibits PDAC cell proliferation and growth both in vitro and in vivo. a Clone formation assay showed that knockdown of LINC01197 dramatically increased HPNE clone numbers. b Clone formation ability of PDAC cell lines, AsPC1, BxPC3, and PANC1, was significantly decreased when LINC01197 was overexpressed. c Xenograft tumor formation assays showed that increased expression of LINC01197 inhibits subcutaneous tumor formation in PANC1 and BxPC3 cell lines. d IHC assays show that expression of Ki67 in PANC1 and BxPC3 stable cells induced tumor formation. P < 0.05 indicates a significant difference and all assays were performed in triplicate