Table 1.

Highly cited microneurographic studies by Wallin and colleagues from the 1970s

Study	Citations	Key Finding(s)
Sundlöf and Wallin (1978b)	472	Marked interindividual differences in MSNA burst incidence that were not linked to resting BP; MSNA linked to spontaneous changes in diastolic blood pressure.
Sundlöf and Wallin (1977)	412	High intraindividual reliability of MSNA across laboratory sessions; simultaneous nerve recordings similar across two limbs.
Hagbarth et al. (1972)	362	Sympathetic ganglionic blocking agents and Lidocaine abolished SSNA; lack of pulse synchrony in SSNA recordings.
Delius et al. (1972c)	360	Mental stress, body cooling, and respiratory movements increased SSNA; maneuvers to elicit baroreceptor did not alter SSNA.
Delius et al. (1972b)	347	Maneuvers that caused forearm or calf vasoconstriction were associated with increases in MSNA; inverse changes of BP and MSNA were observed during Valsalva’s maneuver and mental stress.
Delius et al. (1972a)	331	Pulse rhythmicity of MSNA that was associated with transient reductions in BP; sympathetic ganglionic blockade abolished MSNA.
Sundlöf and Wallin (1978a)	208	Robust increase of MSNA with lower body negative pressure.
Burke et al. (1977)	192	Changes in posture modify MSNA; first recorded observation of quiescent MSNA during syncopal episode.
Wallin and Sundlöf (1979)	165	Taking age into consideration, there was no significant difference of resting MSNA in normotensive and hypertensive subjects.
Wallin et al. (1975)	144	Carotid sinus nerve stimulation inhibited MSNA but not SSNA.
Wallin et al. (1973)	131	No differences in MSNA or SSNA in normotensive and hypertensive subjects; wide variability of MSNA responsiveness to mental stress.

SSNA, skin sympathetic nerve activity; MSNA, muscle sympathetic nerve activity; BP, blood pressure.