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PTHrP (1–34) leads to NOX-dependent ROS production, Src activation, and subsequent EGFR transactivation. Downstream of Akt S473 phosphorylation and MEK/ERK1/2 activation requires EGFR-mediated PI3K activation. Akt and ERK1/2 possibly execute parallel signaling pathways that increase fibronectin and TGF-β expression.
