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. 2019 Apr 10;13:1145–1153. doi: 10.2147/DDDT.S196319

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© 2019 Chen et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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Combination of emodin with PTX inhibited Akt and ERK pathways in A549 cells. A549 cells were treated with 4 μM PTX or/and 10 μM emodin for 72 hr. (A) The expressions of p-Akt and p-ERK in A549 cells were analyzed by Western blotting. (B) The relative expression of p-Akt was quantified by normalizing to Akt. (C) The relative expression of p-ERK was quantified by normalizing to ERK. *P< 0.05 compared with control group. ^##P< 0.01 compared with 4 μM PTX group. Each group performed at least three independent experiments.

Abbreviation: PTX, paclitaxel.