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. 2019 Apr 24;13:373. doi: 10.3389/fnins.2019.00373

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Copyright © 2019 Dreier, Major, Lemale, Kola, Reiffurth, Schoknecht, Hecht, Hartings and Woitzik.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Terminal SD in the wake of circulatory arrest. Sudden circulatory arrest was induced in this rat by injection of 10 ml of air into the heart via the femoral vein. MAP fell rapidly from 99 to 6 mmHg and respiratory fluctuations of expiratory pCO₂ ceased within 18 s; non-spreading depression caused isoelectricity within 26 s (asterisks) and terminal SD started after 98 s. Terminal SD showed an initial and a late negative DC component similar to human recordings (Dreier et al., 2018b). Whereas the initial one is the actual SD component, the late one is termed the NUP.