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. 2016 May 3;22(8):670–676. doi: 10.1111/cns.12560

Figure 3.

Figure 3

MSC‐CM mitigates the deterioration of neurotransmission in spinal motor neuronal axons of SCA1‐KI mice. Latency to induce the contraction of the hind gastrocnemius muscle following electrical stimulation over the fourth lumbar vertebra was measured. (A) Representative CMAPs recorded in naïve WT (upper) and control and MSC‐CM‐injected SCA1‐KI mice (lower) are shown. (B) The mean latencies of the CMAPs were quantified and compared in control (n = 10 from 5 mice) and MSC‐CM‐treated (n = 12 from 6 mice) SCA1‐KI mice. The error bars represent the SEM. **P < 0.01.