Skip to main content
. 2014 Jan 31;20(5):411–419. doi: 10.1111/cns.12228

Figure 3.

Figure 3

NaHS facilitates the action potential firing and the neuron network activity. (A) Representative traces showing the firing of action potentials by successively greater depolarizing current injections (110, 150, and 200 pA/3.5 s) in control and 200 μM NaHS‐treated groups. (B) The number of action potential firing was increased in NaHS‐treated group (control: 5.9 ± 1.8, NaHS: 14.2 ± 2.5, 110 pA/3.5 s; control: 14.2 ± 2.6, NaHS: 21.7 ± 2.7, 150 pA/3.5 s; control: 22.4 ± 3.7, NaHS: 30.8 ± 3.4, 200 pA/3.5 s). n = 10, paired t‐test, **P < 0.01 versus control. (C) Representative sEPSC traces in control and NaHS‐treated slices. (D, E) The amplitude of sEPSC was increased in NaHS‐treated group (n = 6, P < 0.01). (F, G) The interval of sEPSC was decreased in NaHS‐treated group (n = 6, P < 0.01). (H) Representative mEPSC traces in control and NaHS‐treated slices. (I, J) The amplitude of mEPSC was increased in NaHS‐treated group (n = 5, P < 0.05). (K, L) The interval time of mEPSC was not obviously increased in NaHS‐treated group (n = 5). *P < 0.05; **P < 0.01 versus control; Kolmogorov–Smirnov test.