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. 2019 Apr 26;57(5):e01664-18. doi: 10.1128/JCM.01664-18

TABLE 3.

Distribution of pulsotypes by locale and source among 133 isolates of E. coli ST1193

Pulsotypea No. of isolates
Total (n = 133) United States
Australian clinicalc
Fecalb MVAMC, 2014–2018
Clinicalb,c
Human (n = 28) Dog (n = 1) MVAMC, 2010–2018 (n = 68) 24 VAMCs, 2011 (n = 4) Olmsted County, MN, 2011 (n = 2) UMMC, 2013 (n = 4) Human, 2008 (n = 24) Dog, 2008 (n = 2)
1297 56 14 1 26 2 2 3 8 0
2123 29 6 0 23 0 0 0 0 0
1298 18 1 0 2 0 0 0 14 1
2361 10 3 0 5 0 0 1 0 1
2359 4 2 0 1 0 0 0 1 0
2364 4 2 0 2 0 0 0 0 0
2362 3 0 0 2 1 0 0 0 0
2365 3 0 0 2 1 0 0 0 0
a

Of 13 total ST1193 pulsotypes, 8 (shown in the table) accounted for ≥3 isolates each (127 total isolates; 95% of 133). The remaining 5 ST1193 pulsotypes accounted for either 2 isolates (pulsotype 2366; 2 MVAMC clinical isolates) or 1 isolate (3 MVAMC clinical isolates, 1 Australian human isolate) each.

b

Among U.S. isolates, all prevalence comparisons between fecal and clinical isolates for individual pulsotypes, P > 0.50 (Fisher’s exact test).

c

Among clinical isolates, the U.S. and Australian isolates differed significantly for the prevalence of pulsotypes 2123 (23/68 [28%] [United States] versus 0/26 [0%] [Australian]; P < 0.001) and 1298 (2/68 [3.0%] [United States] versus 15/26 [58%] [Australian]; P < 0.001). For all other pulsotypes, P was >0.05.