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. 2019 Apr 22;2019:7940808. doi: 10.1155/2019/7940808

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Copyright © 2019 Shao-Yang Zhao et al.

This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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ICA regulated the biological activity of Bax. (a) ICA inhibited Bax transfer to mitochondria. (b) Bax dimer was detected by chemical cross-linking technology. (c and d) The reverse effects of Bax gene silencing on ICA-mediated antioxidative stress and neuroprotection were detected using MitoSOX and MTT assay. Scale bar = 50 μm. Data were presented as mean ± S.D. from independent experiments performed in triplicate. ^###P < 0.001 relative to the control group; ^∗P < 0.05 relative to the model group. N.S., not significant.