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. 2019 Jan 14;151(5):670–679. doi: 10.1085/jgp.201812214

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© 2019 Gregorich et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms/). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 4.0 International license, as described at https://creativecommons.org/licenses/by-nc-sa/4.0/).

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Figure 2. — Ablation of ENH protein expression slows tension redevelopment kinetics in mouse myocardium. (A and B) Representative tension redevelopment traces at different [Ca²⁺]_free in skinned right ventricular trabeculae isolated from the hearts of ENH^+/+ (A) and ENH^−/− (B) mice. (C) k_tr–pCa relationships established in ENH^+/+ and ENH^−/− trabeculae. (D) k_tr–relative tension relationship established in ENH^+/+ and ENH^−/− trabeculae. (E) In(k_tr)–relative tension relationships established in ENH^+/+ and ENH^−/− trabeculae. The natural logarithm of mean k_tr values were plotted against relative tension, and the regression fitted to the data yielded y intercepts of 1.60 (g_app = 4.94 s⁻¹) and 1.14 (g_app = 3.33 s⁻¹) for ENH^+/+ and ENH^−/− trabeculae, respectively. Each data point is the mean and error bar the SEM.