Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2019 Apr 18;17(6):5581–5589. doi: 10.3892/ol.2019.10274

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright: © Huang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

PMC Copyright notice

Figure 4. — Scue attenuates Alzheimer's disease-like pathology in rats. (A) Latency of crossings and (B) average number of crossings in the Morris water maze spatial probe test of memory. Hippocampal expression of (C) Aβ, (D) SOD, (E) MDA and (F) Ach. (G) Nissl staining in the hippocampus. Scale bar, 50 µm. The expression levels of (H) Bcl-2, Bax and cleaved-caspase-3 were detected by western blotting. Densitometry analyses were conducted using β-actin as an internal reference. Representative results from repeated experiments are presented and data are presented as the mean ± standard deviation. n=6. **P<0.01, ***P<0.001 vs. the control group; ^#P<0.05, ^##P<0.01, ^###P<0.001 vs. the Aβ group. ^{^}P<0.05, ^{^^}P<0.01 vs. the Scu group. Aβ, amyloid β; Scu, scutellarin; Scue, scutellarein; SOD, superoxide dismutase; MDA, malondialdehyde; Ach, acetylcholine; Bcl-2, B cell lymphoma-2; Bax, apoptosis regulator BAX.