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. 2019 Mar 19;17(6):4907–4913. doi: 10.3892/ol.2019.10163

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Copyright: © Lin et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — KMT5A improves ccRCC cell migration and invasion. Representative Transwell migration assay images and quantification of migratory cells for renal call carcinoma (A) 786-O and (B) ACHN cells, which had been transfected with SCR sequence, had the KMT5A expression silenced with shKMT5A, and had the KMT5A expression rescued with shKMT5A-RES. Representative Transwell invasion assay images and quantification of invasive cells for renal call carcinoma (C) 786-O and (D) ACHN cells, which had been transfected with SCR sequence, had the KMT5A expression silenced with shKMT5A, and had the KMT5A expression rescued with shKMT5A-RES. Significant differences were confirmed by one-way analysis of variance. **P<0.01, shKMT5A group compared with the SCR group. KMT5A, N-lysine methyltransferase KMT5A; SCR, scrambled control; shKMT5A, short hairpin RNA targeting KMT5A, shKMT5A-RES.