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SA disrupts the ABA‐induced interaction between PP2Cs and PYL1. (a–c) Sensorgrams obtained with recombinant, purified 10 μM of His6‐tagged PP2C‐D4 (a), 1 μM of ABI1 (b) or 2.5 μM of ABI2 (c) using a His6‐SUMO‐tagged PYL1‐immobilized sensor chip in the absence or presence of the indicated concentrations of ABA or SA. Signals detected from a mock‐coupled control chip were subtracted. The experiments were independently repeated at least twice
