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. 2019 Jan 8;33(2):221–230. doi: 10.1007/s10557-018-6845-3

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© The Author(s) 2019

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 4 — The accumulation of cMyC vs. cTnT after myocardial injury caused by intracoronary ethanol. Venous blood was collected frequently over the first 2 h, and up to 24 h, after therapeutic alcohol septal ablation for hypertrophic cardiomyopathy (TASH) using ethanol infused selectively into a septal perforating branch coronary artery. Summary data of absolute quantification of cMyC (open symbols) vs. cTnT (closed symbols) over time following TASH (n = 20). Inset figure is a zoom of the first 240 min. Over this time interval, cMyC accumulates in the serum approximately six times faster than cTnT (slope 25.8 ± 1.9 vs. 4.0 ± 0.4 ng/L/min, p < 0.0001). Figure reproduced from Baker et al. [22]