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. 2019 Apr 23;17(6):4457–4464. doi: 10.3892/etm.2019.7522

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Copyright: © Liu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — TGF-β overexpression enhances the activation of ERK/MAPK in ESCs. (A) Representative immunoblots and quantitative analysis of (B) ERK phosphorylation, (C) p38 and (D) JNK expression in ESCs following transient transfection of TGF-β plasmid or empty vector. Data are presented as the mean ± standard error of the mean, and this procedure was performed in triplicate. **P<0.01 vs. vector group. TGF, transforming growth factor; ERK, extracellular signal-regulated kinase; MAPK, mitogen-activated protein kinase; ESC, endometrial stromal cell; JNK, c-Jun N-terminal kinase; p, phosphorylated.