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. 2019 May 10;20:88. doi: 10.1186/s12931-019-1063-5

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© The Author(s). 2019

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Representative microscopy images for a) immunohistochemical 8-oxodG staining to detect oxidative DNA damage in pulmonary sections of hyperoxia and caffeine exposed rats. Quantitation of 8-oxodG cells counts in lung sections at b) P3 and with recovery on P15 (P3_P15) and P5 and with recovery on P15 (P5_P15), respectively. Data are expressed relative to the normoxia-exposed control group (100%) and the 100% values are 13.3 (P3), 7.0 (P3_P15), 6.2 (P5), and 7.6 (P5_P15) cells per mm^− 2, respectively. Error bars represent SEM, n = 6–8/group. **p < 0.01 and ***p < 0.001 versus control (NO); ###p < 0.001 versus hyperoxia group (HY) with Mann Whitney test