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. 2019 May 7;12:3509–3517. doi: 10.2147/OTT.S195404

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© 2019 Han et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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miR-506-3p inhibition restored the si-HOXA11-AS-induced effects on cell proliferation, cycle arrest and apoptosis in RB cells. (A) The relative expression of miR-506-3p expression was determined in Y79 cells transfected with si-NC or si-HOXA11-AS, with or without the miR-506-3p inhibitor. (B–D) Cell proliferation, cycle arrest and apoptosis were determined in Y79 cells transfected with si-NC or si-HOXA11-AS, with or without the miR-506-3p inhibitor. *P<0.05, **P<0.01.

Abbreviation: RB, retinoblastoma.