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Exenatide ameliorated advanced glycation end products (AGEs)-induced degradation of aggrecan in human primary chondrocytes. Human primary chondrocytes were stimulated with 100 μg/ml AGEs in the presence or absence of exendin-4 (a form of exenatide) at the concentrations of 10 and 20 nM for 48 h. Expression of aggrecan was determined (a, b, c, P < 0.01 vs. previous column group).
