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. 2019 May 1;8(9):e012016. doi: 10.1161/JAHA.119.012016

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© 2019 The Authors. Published on behalf of the American Heart Association, Inc., by Wiley.

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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A, Urine Na⁺ excretion (U_N _aV) under the following conditions: (clear bar) female Wistar‐Kyoto (WKY) right (RT) kidney renal interstitial (RI) control infusion (N=12), (striped bar) female WKY left (LT) kidney RI angiotensin III (Ang III) (3.5, 7.0, 14.0, and 28.0 nmol/kg per min) infusion (N=6), (black bar) female WKY LT kidney RI Ang III+PD (10 μg/kg per min) infusion (N=6), (gray bar) female spontaneously hypertensive (SHR) RT kidney RI control infusion (N=7), (striped bar) female SHR LT kidney RI Ang III infusion (N=7), all in the presence of systemic AT ₁R blockade. B, Mean arterial pressure (MAP) under conditions described in A. Data represent mean±1 SE. *P<0.05 and **P<0.01 from own control and ⁺ P<0.05 and ⁺⁺ P<0.01 from corresponding Ang III+PD periods.