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. 2019 May 8;9:142. doi: 10.3389/fcimb.2019.00142

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Copyright © 2019 Wang, Li, Ma, Tang, Tang, Hu and Liu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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AvrA increases the tolerances to oxidative stress and iron deficiency. (A) The secondary structure prediction of AvrA by software RNAfold. (B) Growth curves of wild type, ΔavrA and the complemented strain in iron deficiency treatment. (C) The survivals of wild type, ΔavrA and the complemented strain under H₂O₂ stress in the concentrations ranging from 0 to 10 mM (D) Relative expression of iscR operon at stationary phage in iron deficiency treating with DIP. ^**p < 0.01, following by one-way ANOVA, independent t-tests.