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. 2019 May 14;20:372. doi: 10.1186/s12864-019-5765-3

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Orthologous relationship of 1519 mammal obesity genes. a Orthologous relationship of obesity genes between goose and chicken or goose and human. b Proportion of DEGs in each relationship category. Numbers marked on the bar show the total number of gene families in that category. Most of these genes shared a 1:1 orthologous relationship to human (74.66%) and more genes were 1:1 orthologous to chicken genes (80.53%). Some gene families showed either contraction (7.16 and 5.34% to human and chicken, respectively) or expansion (1.96 and 8.08% to human and chicken, respectively). Differentially expressed genes identified in liver also showed strong enrichment among these obesity genes (Chi-square p-value = 4.42*10^− 4). Most DEGs shared a 1:1 orthologous relationship with human (or chicken) and accounted for 18% of all goose-human 1:1 obesity genes (or 18.49% of goose-chicken 1:1 obesity genes)