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Potential mechanism of type 2 inflammation in CRS patients. S. aureus, Staphylococcus aureus; IL-, Interleukin; TSLP, Thymic Stromal Lymphopoietin; TSLPR, TSLP Receptor; Th2, T helper 2 cell; ILC2, Innate-like cell 2; IgE, Immunoglobulin E; SE, S. aureus enterotoxin; mDC, Myeloid Dendritic cell.
