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. 2019 Apr 5;55(4):95. doi: 10.3390/medicina55040095

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© 2019 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Potential mechanisms of non-type 2 inflammation in CRS patients. TLR, Toll-like Receptor; PAMP, Pathogen Associated Molecule; IFN-γ, Interferon gamma; IL-, Interleukin; MPO, Myeloperoxidase; TNFα, Tumor Necrosis Factor alpha; Th1, T helper 1 cell; Th17 T helper 17 cell; Th22, T helper 22 cell; Treg, regulatory T cell; TGF-β Transforming Growth Factor.