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. 2019 May 8;160(9):2136–2148. doi: 10.1097/j.pain.0000000000001607

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Copyright © 2019 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of the International Association for the Study of Pain.

This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal.

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Figure 6. — Translocator protein-18 kDa colocalizes with activated microglia in the spinal cord at 7 days after fracture. Images of control and CRPS spinal cord tissue show extensive colocalization of TSPO in activated microglia (CD11b+) at 7 days with only low levels of colocalization at baseline or 7 weeks after fracture. Scale bars are 50 µm (×20) and 50 µm (×60). White arrows in ×60 images indicate cells that are magnified in right hand panels from region outlined by white box in ×20 overlay. Representative images are shown from n = 5 control, n = 5 CRPS 7 days and n = 6 CRPS 7 weeks mice, with 3 to 5 spinal cord sections examined per mouse. CRPS, complex regional pain syndrome.