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. 2019 May 21;10(6):398. doi: 10.1038/s41419-019-1628-8

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a (Left) Schematic diagram of HR reporter system. (Right) Analysis of HR repair activity in MDA-MB-231 cells treated with 50 nM of perifosine. b–e Analysis of RAD51 foci (b), BRCA1 foci (c), BRCA2 foci (d), or RPA2 foci (e) in MDA-MB-231 cells after exposure to indicated treatment for 24 h. f Perifosine did not change cell cycle distribution of MDA-MB-231 cells. **P < 0.01, n.s. no significance