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. 2019 May 22;18:117. doi: 10.1186/s12944-019-1066-8

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© The Author(s). 2019

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — ApoJ attenuated the ox-LDL-stimulated ROS production. a. Nox2/gp91^phox expression in NRVCs, as determined by western blotting. Ox-LDL enhanced Nox2/gp91^phox expression in control adenovirus-infected cells, which was attenuated by ApoJ-overexpression (n = 4/group). b. Nox2/p47^phox expression, as determined by western blotting (n = 4/group). Ox-LDL increased Nox2/p47^phox expression in control adenovirus-infected NRVCs, while ApoJ overexpression prevented Nox2/p47^phox expression. ^*P < 0.05 versus control Ad/PBS group, ^**P < 0.01 versus control Ad/PBS group; ^#P < 0.05 versus control Ad/ox-LDL group, ^##P < 0.01 versus control Ad/ox-LDL group