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. Author manuscript; available in PMC: 2020 May 1.
Published in final edited form as: Exp Neurol. 2019 Jan 31;315:32–41. doi: 10.1016/j.expneurol.2019.01.019

Fig. 5.

Fig. 5.

A) Mean (± SEM) cortical lesion volume (mm3) 5 weeks after controlled cortical impact injury at 2.8 cortical deformation depth. Left to right: TBI + VEH group represents rats that were injured and did not receive drug treatment; TBI + GAL (1 mg/kg) and TBI + GAL (2 mg/kg) groups represent rats receiving one of the two doses of GAL administration. CCI injury produced the largest cortical lesion volume, with GAL dose-dependently reducing the extent of cavity formation (*p<0.01 versus TBI + VEH group). B-D) Panels are mean depictions of the lesion within each group at the level of the dorsal hippocampus (TBI + VEH at top, TBI + GAL (1mg/kg) in middle, TBI + GAL (2 mg/kg) at bottom).