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. Author manuscript; available in PMC: 2019 May 28.
Published in final edited form as: Nat Microbiol. 2018 Mar 27;3(4):503–513. doi: 10.1038/s41564-018-0131-9

Fig. 2 |. HCMV NR-1 infection reprogrammes human CD34+ HPCs into a long-life monocyte subset at the late stage of infection.

Fig. 2 |

a, Expression of surface markers on conventional mature monocytes and HPCs infected with NR-1 or Mock at 14 dpi. BM, bone marrow. b,c, NR-1-infected HPCs at 14 dpi displayed delayed apoptosis (b) and increased cell viability (c) compared to mature monocytes and Mock-infected HPCs. d, Cytokine level in mature monocytes and HPCs infected with NR-1 or Mock at 14 dpi. Data are presented as the mean ± s.e.m. of three independent experiments. *P < 0.05, **P < 0.01 as determined by the two-tailed t-test (the P values are detailed in Supplementary Table 1).