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. 2019 May 13;20(9):2369. doi: 10.3390/ijms20092369

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© 2019 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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KT treatment improves hepatic lipid metabolism in MCD-fed db/db mice (a) qRT-PCR analysis for cluster of differentiation 36 (Cd36), peroxisome proliferator-activated receptor gamma (Pparγ), Diacylglycerol-O-acyltransferase 2 (Dgat2), fatty acid synthase (Fas), sterol regulatory element binding protein 1c (Srebp1c), peroxisome proliferator-activated receptor gamma coactivator 1 alpha (Ppargc1α), fatty acid binding protein (Fabp1), acyl coenzyme A oxidase 1 (Acox1), carnitine palmitoyltransferase 1 (Cpt1), peroxisome proliferator-activated receptor alpha (Pparα), and medium-chain-acyl-coenzyme A dehydrogenase (Mcad) in livers of the experimental animals. Results were described as fold increase. (b) Hepatic level of free fatty acids in these mice. All of the results are display as mean ± SEM (n ≥ 3/group, * p < 0.05, ** p < 0.005 vs. control group).