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. 2019 May 24;10:547. doi: 10.3389/fneur.2019.00547

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Copyright © 2019 Xu, Chen, Zhao, Chen and Zhang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Brain magnetic resonance imaging (MRI) of a patient with cerebral amyloid angiopathy (CAA)-related inflammation (CAA-RI) with the ApoE ε2/ε2 genotype. Before glucocorticoid treatment, axial fluid attenuated inversion-recovery (FLAIR) (A–D) sequences revealing multiple, asymmetrical subcortical white matter lesions, predominantly in the left lobe. The lesions are isointense on diffusion-weighted imaging (DWI) sequence (E), hyperintense on apparent diffusion coefficient (ADC) (F) and without parenchymal enhancement (G). MRA did not reveal abnormalities (H). Scattered hypointense lacunae (arrow head) and iso-hyperintense lesions (arrow) on the DWI sequence indicating microbleeds and subacute ischemia. On the follow-up MRI 2 months later, FLAIR lesions largely disappeared after glucocorticoid treatment (I–L). Susceptibility weighted imaging revealed no changes in diffuse microbleeds (M–P).