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. 2019 Jun 4;51(6):63. doi: 10.1038/s12276-019-0255-x

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 9 — Exosomal miR-328 in M2 macrophages regulates PF progression via FAM13A. miR-328 exhibited upregulated expression in rats with PF and was carried by M2 macrophage-derived exosomes. M2 macrophage-derived exosomes with silenced miR-328 conferred resistance to pulmonary interstitial fibroblast proliferation and inhibited PF by directly upregulating FAM13A expression