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. 2019 Jun 5;10:2452. doi: 10.1038/s41467-019-10279-w

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Molecular mechanism of compound binding to the membrane cavity. a Details of EBP binding to U18666A. The hydrophilic interactions are indicated by dashed lines. The residues related to substrate recognition are shown as sticks. b Details of EBP binding to tamoxifen, annotated as in a. c Overall view of the membrane cavity. d Binding of EBP mutants to [³H]-Ifenprodil. Data shown are the mean ± SD of triple times determinations. Source data are provided as a Source Data file