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Silencing ARID1A was correlated with Akt pathway activation in NPC cells and clinical NPC biopsies. (A) Depletion of endogenous ARID1A by siRNA increased the phosphorylation of Akt in CNE1 and HNE1 cells. (B and C) Representative images revealed that low ARID1A expression levels were significantly associated with high p-Akt levels in NPC biopsies. #P<0.001.
Abbreviations: NPC, nasopharyngeal carcinoma; ARID1A, AT-rich interacting domain-containing protein 1A.
