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. 2019 Jun 6;10(6):448. doi: 10.1038/s41419-019-1671-5

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a Binding sites of miR-708 at SPHK2 3′-untranslated region (3′-UTR), SPHK2 wild-type (WT), and mutant (MT) 3′-UTR are indicated. b The luciferase activity in SPHK2 wild-type construct, instead of luciferase activity in mutant construct, was decreased when miR-708 was presented. c miR-708 decreased SPHK2 messenger RNA (mRNA) expression level in LN382 and GBM-GY cells. d miR-708 decreased SPHK2 protein levels in LN382 and GBM-GY cells. e The reversed association between miR-708 and SPHK2 expression was indicated in glioma tissues