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. 2019 May 31;10:660. doi: 10.3389/fphys.2019.00660

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Copyright © 2019 Ge, Wang, Zhang, Li and Hu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Tempol treatment inhibits oxidative stress in acetaminophen-induced acute hepatotoxicity. (A) Levels of SOD in liver from the control mice (control), vehicle-treated hepatotoxicity mice (APAP), and tempol-treated hepatotoxicity mice (APAP + tempol). (B) Levels of catalase in liver from three groups. (C) Levels of H₂O₂ in liver from three groups. (D) Levels of MDA in liver from three groups. (E) Levels of GSH in liver from three groups. Data are mean ± SEM. **p < 0.01 vs. control, ^##p < 0.01 vs. APAP, ***p < 0.001 vs. control, ^###p < 0.001 vs. APAP, n = 6.