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. 2019 May 31;10:533. doi: 10.3389/fphar.2019.00533

Table 3.

PK parameters of EC3039 and EC1728 administered to a satellite group of dogs for 5 days by oral gavage.

Gender Compound detected Cmax (ng/mL)
AUClast (hng/mL)
T1/2 (h)
Day 1 Day 5 Day 1 Day 5 Day 5
EC3039 (1 mg/kg) M EC3039 400 2,300 2,490 72,100 33
EC17281 5 101 26 6,370
F EC3039 568 17,300 3,940 361,000 40
EC1728 4 404 24 25,200
EC3039 (5 mg/kg) M EC3039 16,900 32,100 46,500 463,00 27
EC1728 54 772 295 50,200
F EC3039 18,000 30,900 67,200 589,000 25
EC1728 50 606 298 39,000
EC1728 (1 mg/kg) M EC1728 330 1,900 1,690 32,000 27
F EC1728 308 4,660 1,350 155,000 33
EC1728 (5 mg/kg) M EC1728 1,100 1,930 3,760 96,300 25
F EC1728 1,160 4,990 4,940 53,200 23

EC3039 had improved PK properties compared to EC1728. EC3039 had higher systemic exposure (Cmax and AUC) compared to EC1728, and exposure increased in a more than dose proportional manner for EC3039 and less than dose proportional manner for EC1728. Exposure for both compounds increased over the 5-days of dosing. 1EC1728 was detected in animals treated with EC3039, presumable from N-demethylation by CYP450; however, the T1/2 could not be calculated because it could not be fitted to a PK model.