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CD38 may alter the availability of NAD+ precursors. CD38+ cells may limit the growth of pathogens such as H. aegyptius, H. influenzae, H. haemolyticus, H. parainfluenzae, and H. parahaemolyticus by degrading extracellular NAD+ precursors required for NAD+ synthesis in bacteria. In the absence of NAD and its precursors (V factor), pathogens undergo metabolic collapse.
