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. 2019 Jun 10;9:8401. doi: 10.1038/s41598-019-44835-7

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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MAP3K3 affected human OC cell migration and apoptosis in vitro. Transwell migration and invasion experiment (A,B), cell scratch wound-healing assays (D,E), and flow cytometry (G,H) to detect the effect of MAP3K3 expression on biological function in SKOV3 and A2780 cells. Effect of MAP3K3 overexpression and blocking the NF-κB signalling pathway (MAP3K3 + QNZ) in OV2008 cells on cell migration and invasion (C), cell motility (F), and cell apoptosis (I). Repeated three independent experiments, ^*means p < 0.05, ^**means p < 0.01, ^***means p < 0.001.