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. 2019 Jun 11;9:8460. doi: 10.1038/s41598-019-44906-9

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Reduced oxidative stress marker (8-isoprostane) accumulation in the subarachnoid haematoma of mice with induced neutropenia. (A) Isolation of the basal subarachnoid haematoma (drawing by Stefan Kindel, Mainz, Germany). (B) 8-isoprostane levels in the subarachnoid haematoma after induction of neutropenia or vehicle treatment. Note that the significant increase in the 8-isoprostane levels 3 h after SAH in the vehicle group was markedly attenuated in the neutropenia group. (*p < 0.05; error bars indicate SEM; group sizes: SAH-neutropenia (biochemistry groups 1–3): 15 min n = 6, 3 h n = 5, 24 h n = 5 ; SAH-control (biochemistry groups 4–6): 15 min n = 6; 3 h n = 4; 24 h n = 5).