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. 2019 May 23;18(1):443–450. doi: 10.3892/etm.2019.7599

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Copyright: © Chen et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Immune cell infiltration and lung injury in CLP-induced ALI in WT and TLR^−/− mice. BALF samples were collected from WT and TLR^−/− mice 20-h post-surgery and the (A) total, (B) PMN and (C) macrophage count was examined in each group. (D) BALF protein concentration was examined. (E) Morphological changes were observed following H&E staining in lung tissue sections (magnification, ×200). Scale bar, 200 µm. Data from at least three independent experiments. (F) CLP-induced lung injury scores were examined. Data are presented as the mean ± standard error of the mean (n=5). ^#P<0.05 vs. the WT sham group; **P<0.01 vs. WT CLP group. CLP, cecal ligation and puncture; ALI, acute lung injury; WT, wild-type; TLR, Toll-like receptor; PMN, polymorphonuclear cells; BALF, bronchoalveolar lavage fluid; H&E, hematoxylin and eosin.