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. 2019 May 9;18(1):25–32. doi: 10.3892/etm.2019.7564

Figure 4.

Figure 4.

miR-122 functions as a downstream effector in the UCA1-mediated proliferation and invasion of intrahepatic cholangiocarcinoma cells. UCA1-transfected 9810 cells were transfected with miR-122 or miR-NC mimics. (A) miR-122 was upregulated in the UCA1+miR-122 group when compared with that in the UCA1+miR-NC group. (B) The cell proliferation and (C) invasion were also upregulated in the UCA1+miR-122 group when compared with those in the UCA1+miR-NC group. Magnification, ×200. **P<0.01 vs. UCA1+miR-NC. UCA1, urothelial carcinoma-associated 1; NC, negative control; miR, microRNA; OD, optical density.