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. 2019 Apr 24;18(1):389–396. doi: 10.3892/etm.2019.7527

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Copyright: © Fei et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Inflammatory factors promote miR-18 expression in islet β-cells. (A) miR-18 expression was markedly elevated in cytokine-induced MIN6 cells. (B) miR-18 levels were upregulated by induction with IL-1β, TNF-α and IFN-γ or their combination. (C) The upregulation or knockdown efficacies of miR-18 mimics and inhibitor in MIN6 cells. *P<0.05 vs. Control. miR, microRNA; IL, interleukin; IFN, interferon; TNF, tumor necrosis factor; NC, negative control; Cyt mix, mixture of cytokines IL-1β, TNF-α and IFN-γ.