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. 1997 Apr 1;17(7):2295–2313. doi: 10.1523/JNEUROSCI.17-07-02295.1997

Fig. 9.

Fig. 9.

Anterograde axonal transport of BDNF by afferent systems. Bright-field photomicrographs showing sections processed for BDNF immunohistochemistry through amygdala (A, B), parabrachial region (C), and habenula (D–F) in control rats (A, D) or rats that received either an electrolytic lesion of the lateral parabrachial nucleus (C) or an aspirative lesion of the septofimbrial and triangular nuclei (E, F; lesion site is not shown). As seen in A–C, BDNF immunolabeling in the central nucleus (Ce) of the amygdala (A) is almost completely eliminated (B) after ablation of cell bodies in the lateral parabrachial nucleus (LPB) (C); in C, the lesion destroyed much of the LPB but spared the medial parabrachial nucleus (MPB). D–F show that BDNF-ir in the medial habenular nucleus (MHb) (D) is lost ipsilateral to an unilateral septofimbrial/triangular septal nuclei lesion (E) or bilaterally after bilateral lesions of these fields (F). Scale bars (shown inA): A, B, 500 μm; C, 500 μm; (shown in D): D–F, 750 μm.BLA, Basolateral amygdala; CeL, central amygdaloid nucleus, lateral division; CeM, central amygdaloid nucleus, medial division; La, lateral amygdala; LC, locus coeruleus; LHb, lateral habenular nucleus; opt, optic tract;PV, paraventricular thalamic nucleus.