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. 1996 Sep 1;16(17):5334–5343. doi: 10.1523/JNEUROSCI.16-17-05334.1996

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Copyright © 1996 Society for Neuroscience

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Fig. 3. — Passive propagation of LTP in vertical OAIs is restricted to the second component of the EPSP. Response of the st. pyramidale fPS to pairing and tetanization (A1) shows that the slices are responsive to both 1 Hz (bar) and tetanic (arrow) stimulation (53.5 ± 7.2 and 122.1 ± 17.9% after 1 and 100 Hz, respectively). The analysis of the interneuron EPSP is shown in A2 for the first and inA3 for the second component. Note that changes after the LTP-inducing protocols appear only in the later component (A3) (EPSP₂, 69.2 ± 7.0 and 117.3 ± 13.6% after pairing and tetani, respectively) and not in the early component (A2) (EPSP₁, 99.8 ± 5.2 and 90.9 ± 4.87% after pairing and tetani, respectively). Data fromn = 3 perforated-patch and n = 3 whole-cell experiments. Insets are averaged traces in control (1), after pairing (2), after tetanization (3), and superimposed (1–3). Note also the parallel shift to the left of both the peak fPS and the onset of EPSP₂ after tetanization (arrows in theinsets).