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. 2019 May 9;20(1):33–40. doi: 10.3892/mmr.2019.10215

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Copyright: © Liu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — miR-338-3p inhibits lung cancer development in vivo. (A) Schematic of lung cancer model with nasal cavity treatment of cre-advenovirus (AdCre) in KrasG12D mice. (B) H&E staining showing malignant cell hyperproliferation in AdCre treated tissue. (C) Tumor weights on the 35th day after the primary tumor initiation are decreased in the miR-338-3p treated group compared with the control groups *P<0.05, **P<0.01. (D) AKT-p and total protein level of β-catenin was reduced in the miR-338-3p treated group. (E) Quantification of the relative AKT-p expression levels. Con, control; miR, microRNA; p, phosphorylated; WT, wild-type.